Anti-aging Preventive

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As we age, skin loses its elasticity and becomes thin, weak and prone to wrinkles and hyperpigmentation. The loss of skin thickness due to the lack of collagen support, the aging skin or aged skin looks pale and lacks elasticity. These two properties are the hallmarks of wrinkles and creases. How can this be reversed? The main goal of anti-aging products is to decrease and eventually eliminate the appearance of fine lines and wrinkles and provide even-toning effects. The way to achieve this goal is to prevent skin from further damage, maintaining mainly the integrity of extra cellular matrix (ECM), dermal-epidermal junction (DEJ) areas, controlling glycation and preserving skin hydration.

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Synoxyl® AZ (INCI: Acetyl Zingerone) Use level: 0.5 to 1%

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The first product in the industry to provide skin protection by three distinct preventative pathways – Quencher, Scavenger (antioxidant) & Selective Chelator; Demonstrated to reduce both UV-induced formation of immediate (iCPDs) & delayed cyclobutane pyrimidine dimers (dCPDs); Protects mitochondria by reducing UV-induced cell & mitochondrial membrane disruption & maintains ATP Synthesis; Excellent NADPH oxidase inhibitor – The first step that controls the oxidative stress cascade; Selective & effective chelator for iron & copper –Reducing iron & copper-induced oxidative stress; Far superior to existing commercial chelators; Clinically shown to elevate skin’s defense against UV-induced skin damage without using sunscreens.

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Sytenol® A (INCI: Bakuchiol) Use level: 0.5 to 1%

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Targeting skin concerns early on can effectively prevent damage to the skin’s surface and improve skin quality. Slowing down the aging process can be achieved by using Sytenol® A due to its broad-spectrum antioxidant property and boosting antioxidant defense to limit direct oxidative damage to skin and also due to its anti-inflammatory and matrix metalloprotease inhibitory activities. It has a strong lipid peroxidation inhibition activity which is 60-foldmore powerful than natural tocopherol. Clinically, skin protection property of Sytenol® A (without using sunscreens) has been demonstrated by the reduction of sun-induced skin erythema.

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Synastol® TC (INCI: Terminalia chebula fruit extract) Use level: 0.5 to 1%

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The first natural solution to glycation – works by reversing & inhibiting protein glycation; Glycation affects every types of cells & molecules; Scavenging all five major radicals & non-radical; Inhibiting xanthine oxidase induced superoxide formation; Preserving matrix integrity by inhibiting MMP-1, MMP-2, MMP-3 & MMP-12 (Elastase) activities; Controlling inflammation by inhibiting COX & LOX enzyme activities; Provides protection against environmental pollution- & blue light- induced skin damage; Ecocert & Cosmos Natural; Globally approved.

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Synovea® HR (INCI: Hexylresorcinol) Use level: 0.5 to 1%

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Synovea® HR (HR) is a highly purified material (>99%, typically ranging from 99.5 to 99.9%) which is essentially free of resorcinol, a known skin irritant and is particularly suitable for topical applications. HR is perhaps one of, if not the, most studied and well-known alkylresorcinol. HR has a long and safe history of human use.

HR provides broad-spectrum multi-targeted skin protection by up-regulating antioxidant defense system - glutathione & glutathione enzymes. HR also has moderate glycation inhibitory activity and has a strong NF-κB inhibitory activity which is 8 to 16-fold more effective than Resveratrol and Curcumin, respectively. Globally approved.

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HydraSynol IDL (INCI: Isosorbide Disunflowerseedate) Use level: 2 to 4%

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HydraSynol IDL, a patented product, prepared by reacting isosorbide (obtained from corn) and fatty acids (from sun flower seed oil) using an eco-friendly process. HydraSynol IDL is novel structurally and a stable version of linoleic acid (LA) and provides all the benefits of LA and more.

HydraSynol IDL defends skin barrier by dual pathways – building antioxidant shield by boosting Small Proline-Rich Proteins (SPRPs) and by inhibiting up-stream pro-inflammatory mediators, demonstrated by ex-vivo study.

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